Abstract
Preterm delivery of low birth weight infants (PLBW) remains a significant public health issue and a leadingcause of neonatal death and long-term neurodevelopmental disturbances and health problems. Recent epidemiological and microbiological-immunological studies have siggested that periodontal disease may be an independent risk factor for PLBW. Postulated mechanisms include translocation of periodontal pathogens to the fetoplacental unit and action of a periodontal reservoir of lipopolysaccharides or inflammatory mediators. However, non-causal explanations for the correlation studies, will be necessary before periodontitis can be considered as a causal factor for PLBW.
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